Experimental vaginal candidasis rat model

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Jump to Main Content. Selections 0 Show Selections Clear Selections. Resistance among isolates to azole antifungal agents has been reported.

In vivo activity of Sapindus saponaria against azole-susceptible and -resistant human vaginal Candida species. VVC was induced in hyperestrogenic Wistar rats with azole-susceptible C. The rats were treated intravaginally with 0. The toxicity was evaluated in cervical cells of the HeLa cell line.

Evaluation of efficacy, pharmacokinetics and tolerability of peptidomimetic aspartic proteinase inhibitors as cream formulation in experimental vaginal candidiasis. In vivo studies were also conducted in female albino rats and rabbits to obtain information about the safety, local tolerability and principal pharmacokinetics parameters of the two compounds. Importantly, the two compounds showed marked acceleration of fungus clearance in the rats challenged with the fluconazole-resistant as well as with the capsofungin-resistant strain of C.

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Th17 cells play a major role in coordinating the host defence in oropharyngeal candidiasis. In this study we investigated the involvement of the Th17 response in an animal model of vulvovaginal candidiasis VVC. This production was strongly reduced when Th17 differentiation was inhibited and was increased by rIL treatment.

Metrics details. Vulvovaginal candidiasis VVC is a common infectious disease of the lower genital tract. Nystatin, a polyene fungicidal antibiotic, is used as a topical antifungal agent for VVC treatment.

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Skip to search form Skip to main content. Batista Vulvovaginal candidiasis VVC is regarded as an important public health issue, and several aspects of its pathogenesis are not yet sufficiently clear. Experimental in vivo models of vaginal infection with Candida albicans have been extremely useful in the identification of factors concerning hormonal influences on the infection, the virulence of the yeasts, the susceptibility, and the treatment of the infection.

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Wozniak, K. Protective host defense mechanisms against vaginal Candida albicans infections are poorly understood. Although cell-mediated immunity CMI is the predominant host defense mechanism against most mucosal Candida infections, the role of CMI against vaginal candidiasis is uncertain, both in humans and in an experimental mouse model.

Vulvovaginal candidiasis is a common fungal infection afflicting women which is primarily caused by the yeast Candida albicans C. It is imperative to introduce new drug classes to counter this threat due to the continuous emergence of drug-resistant cases in recent years. The purpose of this study was to clarify the in vivo antifungal activity of perillaldehyde PAE against C.

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